Heart Damage Treatments Annapolis MD

During a heart attack, vessels that supply blood to the heart become blocked, preventing enough oxygen from getting through. The heart muscle dies or is permanently damaged.

Mark Charles Haigney, MD
Annapolis, MD
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Cardiology
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Medical School: Vanderbilt Univ Sch Of Med, Nashville Tn 37232
Graduation Year: 1987

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Louis K Essandoh
(410) 897-9474
888 Bestgate Rd
Annapolis, MD
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Cardiology, Internal Medicine, Cardiovascular Disease

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Barbara A Hutchinson
(410) 573-9805
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Annapolis, MD
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Cardiology, Internal Medicine, Cardiovascular Disease

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Lawrence David Jacobs, MD
(773) 702-1757
19 Thompson St
Annapolis, MD
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Cardiology
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Medical School: Georgetown Univ Sch Of Med, Washington Dc 20007
Graduation Year: 1998

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Barbara Travis Furlow, MD
(410) 224-0010
104 Ridgely Ave Ste 201
Annapolis, MD
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Cardiology
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Medical School: George Washington Univ Sch Of Med & Hlth Sci, Washington Dc 20037
Graduation Year: 1971

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Dennis Mc Coy Hall, MD
(410) 573-9805
888 Bestgate Rd Ste 215
Annapolis, MD
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Medical School: Univ Of Va Sch Of Med, Charlottesville Va 22908
Graduation Year: 1985

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William Clarence Maxted, MD
(410) 573-6480
2002 Medical Pkwy Ste 500
Annapolis, MD
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Medical School: Georgetown Univ Sch Of Med, Washington Dc 20007
Graduation Year: 1992

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Salvatore S Lauria
(410) 224-1495
128 Lubrono Dr
Annapolis, MD
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Cardiology, Cardiovascular Disease

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Kelley W Sullivan
(410) 573-6480
2002 Medical Pkwy
Annapolis, MD
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Cardiology, Cardiovascular Disease

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Dr.Sven Ender
888 Bestgate Rd # 215
Annapolis, MD
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Heart Damage Treatments

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Doctors have been unable to help injured heart tissue renew itself after a heart attack -- until now.

During a heart attack, vessels that supply blood to the heart become blocked, preventing enough oxygen from getting through. The heart muscle dies or is permanently damaged.

But researchers at Children's Hospital Boston report progress toward someday being ale to regenerate heart tissue after a heart attack or heart failure and even in children who are born with congenital heart defects.

In a study on mice, they showed that neuregulin 1 (NRG1), a growth factor involved in the development of the heart and nervous system, can fuel heart-muscle growth and recovery of cardiac function when injected after a heart attack.

This is a significant development because coronary heart disease, which causes heart attack and angina, is the leading cause of death in America.

After birth, heart-muscle cells stop dividing and proliferating. But experts, led by Dr. Bernhard Kuhn and Kevin Bersell of the cardiology department at Children's, restarted the cell cycle with NRG1, spurring the heart-muscle cells to divide and make copies of themselves.

When the team injected NRG1 into live mice once a day for three months after the animals had heart attacks, heart regeneration increased and the pumping function improved, compared with untreated mice.

In addition, the NRG1-injected mice did not show some common aftereffects of heart failure.

The study, funded by the cardiology department at Children's Hospital Boston, the Charles Hood Foundation and the American Heart Association, found that cell growth does not have to come from stem cells. A report on the research appears in the July 24 issue of Cell.

"Although many efforts have focused on stem cell-based strategies, our work suggests that stem cells aren't required and that stimulating differentiated cardiomyocytes [heart-muscle cells] to proliferate may be a viable alternative," Kuhn, the study's senior investigator, said in a news release from the hospital.

More information

The American Heart Association has tips on maintaining a healthy heart.

SOURCE: Children's Hospital Boston, news release, July 23, 2009

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