Heart Damage Treatments Frederick MD

During a heart attack, vessels that supply blood to the heart become blocked, preventing enough oxygen from getting through. The heart muscle dies or is permanently damaged.

Michael William Levangie, MD
(301) 694-5900
310 W 9th St
Frederick, MD
Specialties
Cardiology
Gender
Male
Education
Medical School: Univ Of Ma Med Sch, Worcester Ma 01655
Graduation Year: 1989
Hospital
Hospital: Frederick Mem Hosp, Frederick, Md
Group Practice: Cardiology Center

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Chen Yuan Tung, MD
(301) 694-5900
1803 Beech Ct
Frederick, MD
Specialties
Cardiology
Gender
Male
Education
Medical School: Oh State Univ Coll Of Med, Columbus Oh 43210
Graduation Year: 1992

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Shawn Adrian Buki, MD
(301) 694-5900
310 W 9th St
Frederick, MD
Specialties
Cardiology
Gender
Male
Education
Medical School: Umdnj-Robt W Johnson Med Sch, New Brunswick Nj 08901
Graduation Year: 1993

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Michael LeVangie
(301) 694-5900
310 W 9th St
Frederick, MD
Specialty
Cardiology, Cardiovascular Disease

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Jeffrey Nathan Cowen
(301) 663-5790
915 Toll House Ave
Frederick, MD
Specialty
Cardiology, Internal Medicine, Cardiovascular Disease

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Shawn Adrian Buki
(301) 694-5900
310 W 9th St
Frederick, MD
Specialty
Cardiology, Cardiovascular Disease

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Sherman Kahan
(301) 694-9033
915 Toll House Ave
Frederick, MD
Specialty
Cardiovascular Disease

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Chen Yuan Tung
(301) 694-5900
310 W 9th St
Frederick, MD
Specialty
Cardiology, Cardiovascular Disease

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Paul Nicholas Chomiak
(301) 694-5861
501 W 7th St Ste D
Frederick, MD
Specialty
Thoracic Surgery, Vascular Surgery, Cardiac Surgery

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Dr.Kusay Barakat
(301) 694-5900
310 W 9th St # A1
Frederick, MD
Gender
M
Education
Medical School: Univ Of Baghdad, Coll Of Med, Baghdad
Year of Graduation: 1968
Speciality
Cardiologist
General Information
Accepting New Patients: Yes
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2.0, out of 5 based on 1, reviews.

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Heart Damage Treatments

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Doctors have been unable to help injured heart tissue renew itself after a heart attack -- until now.

During a heart attack, vessels that supply blood to the heart become blocked, preventing enough oxygen from getting through. The heart muscle dies or is permanently damaged.

But researchers at Children's Hospital Boston report progress toward someday being ale to regenerate heart tissue after a heart attack or heart failure and even in children who are born with congenital heart defects.

In a study on mice, they showed that neuregulin 1 (NRG1), a growth factor involved in the development of the heart and nervous system, can fuel heart-muscle growth and recovery of cardiac function when injected after a heart attack.

This is a significant development because coronary heart disease, which causes heart attack and angina, is the leading cause of death in America.

After birth, heart-muscle cells stop dividing and proliferating. But experts, led by Dr. Bernhard Kuhn and Kevin Bersell of the cardiology department at Children's, restarted the cell cycle with NRG1, spurring the heart-muscle cells to divide and make copies of themselves.

When the team injected NRG1 into live mice once a day for three months after the animals had heart attacks, heart regeneration increased and the pumping function improved, compared with untreated mice.

In addition, the NRG1-injected mice did not show some common aftereffects of heart failure.

The study, funded by the cardiology department at Children's Hospital Boston, the Charles Hood Foundation and the American Heart Association, found that cell growth does not have to come from stem cells. A report on the research appears in the July 24 issue of Cell.

"Although many efforts have focused on stem cell-based strategies, our work suggests that stem cells aren't required and that stimulating differentiated cardiomyocytes [heart-muscle cells] to proliferate may be a viable alternative," Kuhn, the study's senior investigator, said in a news release from the hospital.

More information

The American Heart Association has tips on maintaining a healthy heart.

SOURCE: Children's Hospital Boston, news release, July 23, 2009

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